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Humphries Vestergaard posted an update 6 months, 4 weeks ago
non-users (OR = 0.27; 95% CI = 0.08-0.96). PSA increased on average 1.20 ng/mL among other current tobacco users vs. non-users. Among older AA men, cigarette smoking history and current tobacco use were positively associated with an increase in PSA levels and current marijuana use were inversely associated with PSA levels. Future work with studies of diverse patient populations with cancer outcomes are needed to assess whether these behavioral characteristics contribute to racial/ ethnic disparities in prostate cancer outcomes. Our study provides novel evidence regarding potential differences in PSA levels among older AA men according to behavioral characteristics.
Although it is widely assumed that men’s sexual desire and interest in casual sex (i.e., sociosexual orientation) are linked to steroid hormone levels, evidence for such associations is mixed.
We tested for both longitudinal and cross-sectional relationships between salivary testosterone, cortisol, reported sexual desire and sociosexuality in a sample of 61 young adult men, each of whom was tested weekly on up to five occasions.
Longitudinal analyses showed no clear relationships between steroid hormones and self-reported sexual desire or sociosexual orientation. Cross-sectional analyses showed no significant associations between average hormone levels and self-reported sexual desire. However, some aspects of sociosexuality, most notably desire for casual sex, were related to men’s average hormone levels. Men with higher average testosterone reported greater desire for casual sex, but only if they also had relatively low average cortisol levels.
Our results support a Dual Hormone account of men’s sociosexuality, in which the
effects of testosterone and cortisol predict the extent of men’s interest in casual sex. However, we did not detect compelling evidence for an association of within-subject hormone shifts and sexual desire or sociosexual orientation.
Our results support a Dual Hormone account of men’s sociosexuality, in which the combined effects of testosterone and cortisol predict the extent of men’s interest in casual sex. However, we did not detect compelling evidence for an association of within-subject hormone shifts and sexual desire or sociosexual orientation.The Yedoma layer, a permafrost layer containing a massive amount of underground ice in the Arctic regions, is reported to be rapidly thawing. In this study, we develop the Permafrost Degradation and Greenhouse gasses Emission Model (PDGEM), which describes the thawing of the Arctic permafrost including the Yedoma layer due to climate change and the greenhouse gas (GHG) emissions. The PDGEM includes the processes by which high-concentration GHGs (CO2 and CH4) contained in the pores of the Yedoma layer are released directly by dynamic degradation, as well as the processes by which GHGs are released by the decomposition of organic matter in the Yedoma layer and other permafrost. Our model simulations show that the total GHG emissions from permafrost degradation in the RCP8.5 scenario was estimated to be 31-63 PgC for CO2 and 1261-2821 TgCH4 for CH4 (68th percentile of the perturbed model simulations, corresponding to a global average surface air temperature change of 0.05-0.11 °C), and 14-28 PgC for CO2 and 618-1341 TgCH4 for CH4 (0.03-0.07 °C) in the RCP2.6 scenario. GHG emissions resulting from the dynamic degradation of the Yedoma layer were estimated to be less than 1% of the total emissions from the permafrost in both scenarios, possibly because of the small area ratio of the Yedoma layer. An advantage of PDGEM is that geographical distributions of GHG emissions can be estimated by combining a state-of-the-art land surface model featuring detailed physical processes with a GHG release model using a simple scheme, enabling us to consider a broad range of uncertainty regarding model parameters. In regions with large GHG emissions due to permafrost thawing, it may be possible to help reduce GHG emissions by taking measures such as restraining land development.
Identity provides a useful conceptual lens for understanding educational inequalities in science, technology, engineering and mathematics (STEM). In this paper, we examine how paying attention to physical and digital ‘materiality’ enriches our understanding of identity work, by going beyond the spoken, written and embodied dimensions of identity performances that currently dominate the area of STEM identity scholarship. We draw on a multimodal ethnographic study with 36 young people aged 11-14 carried out over the course of oneyear at four UK-based informal STEM learning settings. Data collection included a series of interviews, observations and youth-created portfolios focused on STEM experiences. Illustrative case studies of two young men who took part in a community-based digital arts centre are discussed in detail through the theoretical lenses of Judith Butler’s
and Karen Barad’s
.
We argue that physical and digital materiality mattered for the performances of ‘tech identity’ in that (i) the focother.
We conclude the paper by suggesting that accounting for materiality in STEM identity research not only guides researchers in going beyond what participants say and are observed doing (and thus engendering richer insights), but also offers more equitable ways of enacting research. Further, we argue that more needs to be done to support the translation of identity resources across spaces, such as between experiences within informal and online spaces, on the one hand, and formal education, on the other.The complement system is a stakeholder of the innate and adaptive immune system and has evolved as a crucial player of defense with multifaceted biological effects. Activation of three complement pathways leads to consecutive enzyme reactions resulting in complement components (C3 and C5), activation of mast cells and neutrophils by anaphylatoxins (C3a and C5a), the formation of membrane attack complex (MAC) and end up with opsonization. However, the dysregulation of complement cascade leads to unsolicited cytokine storm, inflammation, deterioration of alveolar lining cells, culminating in acquired respiratory destructive syndrome (ARDS). Similar pathogenesis is observed with the middle east respiratory syndrome (MERS), severe acquired respiratory syndrome (SARS), and SARS-CoV-2. Activation of the lectin pathway via mannose-binding lectin associated serine protease 2 (MASP2) is witnessed under discrete viral infections including COVID-19. https://www.selleckchem.com/products/crt-0105446.html Consequently, the spontaneous activation and deposits of complement components were traced in animal models and autopsy of COVID-19 patients.
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